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School of Medicine

Physician Assistant Studies - Program


BA, BS, or BAS (four-year, any major) from a regionally accredited institution of higher learning in the US or Canada is required. Applicants who are applying this year for the 2018 entering class who have yet to complete a bachelor’s degree must graduate no later than Friday, May 3, 2019, order to be eligible to matriculate into the next PA class that will matriculate on Monday, May 6, 2019. Foreign degrees can be accepted, but must be evaluated and meet US equivalency. Additionally, candidates with foreign degrees must complete a minimum of 60 semester credits of coursework (approximately two years) in the United States, including our prerequisite courses.

All prerequisite coursework must be complete at the time you submit your application by the August 1 submission deadline. Coursework must be completed for a letter grade that is published on your official transcript and verified by CASPA.

Summer 2018 coursework : If you hope to complete your minimum required coursework in summer of 2018, there is some risk of missing the secondary deadline of September 4th:

Summer 2018 coursework

You must submit the CASPA application by August 1st, and you must enter a grade for the summer course, even if the course has not yet ended.

Do not send in the transcript from the institution where you are taking the class to CASPA until after your grade has posted.

If the grade posted on the transcript does not match the one you entered on your application, CASPA will not correct it for you. They will notify you and you must change it yourself. Once you have done so, your application must be re-processed.

If re-processing delays your application past Tuesday, September 4th our secondary deadline, your application will be disqualified.

There is no need for repeated coursework to be verified by CASPA: apply as early as possible.

Prerequisite grades and timeframe: We do not accept AP, CLEP, or audited coursework. Pass/Fail prerequisites accepted only from regionally accredited institutions whose pass minimum is B or higher.Additionally, classes from professional schools, including medical school, are not accepted.

Prerequisite grades and timeframe:

Prerequisite courses must be completed from a regionally accredited institution of higher learning in the United States or Canada for a letter grade. Foreign prerequisite coursework is not accepted. All courses must be entered on the CASPA application. Letter grades of less than C (such as C-) are not accepted for prerequisites, no exceptions.

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© 2018 Oxford University Press

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Background: The effects of food restriction on energy metabolism have been under investigation for more than a century. Data obtained are conflicting and research has failed to provide conclusive results.


Objective: The objective of this study was to test the hypothesis that in lean subjects under normal living conditions, short-term starvation leads to an increase in serum concentrations of catecholamines and thus to an increase in resting energy expenditure.


Design: Resting energy expenditure, measured by indirect calorimetry, and hormone and substrate concentrations were measured in 11 healthy, lean subjects on days 1, 2, 3, and 4 of an 84-h starvation period.


Results: Resting energy expenditure increased significantly from 3.97 ± 0.9 kJ/min on day 1 to 4.53 ± 0.9 kJ/min on day 3 ( P < 0.05). The increase in resting energy expenditure was associated with an increase in the norepinephrine concentration from 1716. ± 574 pmol/L on day 1 to 3728 ± 1636 pmol/L on day 4 ( P < 0.05). Serum glucose decreased from 4.9 ± 0.5 to 3.5 ± 0.5 mmol/L ( P < 0.05), whereas insulin did not change significantly.


Conclusions: Resting energy expenditure increases in early starvation, accompanied by an increase in plasma norepinephrine. This increase in norepinephrine seems to be due to a decline in serum glucose and may be the initial signal for metabolic changes in early starvation.

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For more than a century, the effects of food restriction on energy metabolism have been under investigation. Data obtained are conflicting and research has failed to provide conclusive results. As early as 1915, Benedict ( 1 ) reported a decrease in metabolic rate of 20–30%, induced by prolonged starvation. This finding was confirmed by further studies ( 2 ). Reductions in basal metabolic rate have also been reported after only 2–3 d of starvation ( 3 ). Other authors, however, found no decrease or even a moderate increase in resting energy expenditure ( 4 ).

Several impressive changes in substrate metabolism during starvation have been described ( 5 , 6 ). Glucose oxidation decreases markedly during the first day of starvation ( 7 ) and fatty acids are mobilized, leading to an increase in plasma concentrations of fatty acids and ketone bodies and to an increased rate of fat oxidation ( 2 , 6 ). is an open-source project started by GmbH . We have a growing user community and active community contributions. Our mission is to take middleware to the next-level - the IoT.

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